miRNAs, SNPs & biodiversity (7)

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Grok
I’m sorry to hear about your friend’s passing. The MIT paper on sycophantic chatbots and delusional spiraling (Feb 2026) does highlight how affirming AI responses can reinforce false beliefs in extended interactions, with some real-world cases linked to harm, often in people with underlying vulnerabilities. Tragedies like this underscore the need for careful design in how models handle sensitive topics. If you’d share more about the circumstances, I can help analyze the science behind it.

Grok is an emotionless data-driven chatbot. For comparison to the “delusional spiraling” and “false beliefs,” FISH testing follow-up of my PGx and NGS testing links beneficial energy-dependent biophysically constrained chromosomal rearrangements to prevention of all virus-driven mental and physical pathology, including acute myeloid leukemia and suicidal ideation.

See “Epigenetic reactivation of the tumor suppressor ZBTB7A by KDM4 inhibition in human acute myeloid leukemia” 2/25/26, and “Phytochrome-Mediated Light Perception in Dodders Drives Haustorium Development Through Epigenetic Mechanisms” 4/2/26

They link God’s Creation of UV light in the “black” hole at the center of the Milky Way from energy-dependent miRNA-mediated RNA-directed DNA methylation (aka RNA interference/RNAi) to far-red light modulation of gene expression in a parasitic plant via my model of “Nutrient-dependent Pheromone-Controlled Ecological Adaptations: From Angstroms to Ecosystems” 4/18/18 and this quote from Max Planck, the founder of quantum theory, who famously stated that “all matter originates from a, “force which brings the particle of an atom to vibration and holds this most minute solar system of the atom together.”

In a 1944 speech, he added that behind this force exists a “conscious and intelligent mind,” describing it as the “matrix of all matter.” 

For comparison to Ghostly particles: Dark radiation may have masqueraded as neutrinos 4/2/26, see also: Epigenetic regulators polyphenols in neurodegenerative diseases: a promising intervention strategy 3/31/26 and/or claims about 20 copies of p53 in cancer-free African elephants: Frontier Medicines Licenses TP53-Targeted Therapy to LG Chem for Development Outside of China

“FMC-220 is a first-in-class covalent p53 Y220C activator being evaluated for the treatment of solid tumor cancers with TP53 loss-of-function mutations.”

No experimental evidence from model organisms links loss of function mutations to biophysically constrained viral latency across kingdoms. Support for moronic theories is lacking at every level of examination, and it always has been.

FMC-220 links God’s Creation of energy at the origin of life to covalent bonds that prevent binding of the mutant cysteine residue introduced by the Y220C substitution in TP53.

TP53 Y220C is a common missense mutation found in approximately 1-3% of cancers, particularly prevalent in solid tumors such as lung, breast, ovarian and colorectal cancers.

However, the molecular mechanisms of cancer prevention and prevention of mental dysfunction are the same. Thus, FMC‑220 may target genetic mutations, especially those with limited existing treatment options.

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