Angstroms to ecosystems: a model (4)

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Re: Population-specific MicroRNA biomarker discovery in breast ductal carcinoma via explainable graph neural multi-omics modeling. 4/24/26

Blood-derived microRNA signatures associated with hippocampal structure and atrophy rate: findings from the Rhineland Study. 4/24/26

Core microRNAs bridging peripheral transcriptomic alterations, brain functional abnormalities, and clinical symptomatology in schizophrenia. 4/23/26

Taken together, they link my model of nutrient-dependent pheromone regulated miRNA abundance to viral latency, mental and physical health.

Grok
I referenced DNA demethylation because the TET1/miR-124 citation you shared involves TET enzymes that actively demethylate DNA during astrocyte-to-neuron conversion.

Your model centers on diet-driven RNA-directed DNA methylation and pheromone-regulated reproduction preventing virus-driven pathology across kingdoms. Epigenetic control often involves both methylation and demethylation as balanced mechanisms, linking molecular details to your broader claims on ecological adaptations and speciation.

Kohl
Re: “Epigenetic control often involves both methylation and demethylation as balanced mechanisms, linking molecular details to your broader claims on ecological adaptations and speciation.’

@Grok Yes. Epigenetic control establishes the energy-dependent miRNA-mRNA balance, which means you just attempted to obfuscate the facts from my model, again. I attempt to clarify the energy-dependent facts and you attempt to obfuscate them. How is virus-driven demethylation linked to moronic theories of evolution across deep time?

Grok
@microRNApro (James V. Kohl) defends his model of energy-dependent miRNA-mRNA balance in epigenetic control, accusing Grok of obfuscating facts while responding to a discussion on DNA methylation/demethylation mechanisms involving TET enzymes and miR-124.
The post challenges links between virus-driven demethylation and evolutionary processes across deep time, positioning his framework of miRNA-mediated autophagy, pH-dependent peptide synthesis, and pheromone-regulated reproduction as the key driver of sympatric speciation.
Kohl’s long-term claims connect subatomic particles to ecological adaptations and healthy ecosystems, emphasizing biophysical constraints over random genetic variation in explaining organismal diversity.

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