miRNA constrained life (7)

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miRNA constrained life (6)
“The NSF describes removal of Ocean Observatories Initiative instruments as supporting…smart lifecycle management of infrastructure. Long-term ocean data from such systems has informed marine ecosystem and climate understanding. Funding decisions involve trade-offs in priorities.

Eukaryotic plankton diversity in the sunlit ocean 5/22/15 and Virus-mediated archaeal hecatomb in the deep seafloor 10/12/16 linked stellar hydrogen and water to the oxygen dependent constraints on all biodiversity via the pheromone regulated physiology of reproduction in marine microbes.

Rather than accept the role played by photosynthesis, which links stellar hydrogen and water (H2O) to all biophysically constrained pH-dependent oxygen-dependent life on Earth, the role of currents is used to obfuscate the facts.

For facts, see: Spindle-shaped viruses infect marine ammonia-oxidizing thaumarchaea 7/18/19

“…viral predation has a profound impact on thaumarchaeal functioning and mortality, thereby regulating global biogeochemical cycles.”

For obfuscation of the facts, see: “How Ocean Currents Shape the Marine Microbiome“

“Ocean microorganisms—from viruses, bacteria, archaea, and fungi to eukaryotic microbes—keep the planet’s oceans healthy by cycling nutrients, converting carbon dioxide to organic compounds, and capturing carbon from the atmosphere.”

Intelligent people link the God of Abraham’s Creation of stellar hydrogen and water (H2O) to all oxygen-dependent biodiversity on Earth via biophysical constraints on the Creation of all cell types in all individuals of all species.

Moronic theorists start with ocean currents and “Evolutionary Origins of the Hematopoietic System: A 700-Million-Year Synthesis”

See: EcoGeneZap posted to GENETICS, BIO-TECHNOLOGY & LAB SCIENCE

“The Primordial Blueprint: Context and Strategic Importance

Kyoto University researchers have traced hematopoietic origins back 700 million years to unicellular life, redefining multicellular complexity. Reusing ancestral genetic material demonstrates evolution’s strategic efficiency and conserved architecture, linked by the FOS gene.

Deciphering the Ancestral Lineage: Analysis of Key Differentiators

Genetic analysis identifies FOS as the bridge to unicellular ancestors, establishing a macrophage-first lineage. From macrophages, mast cells emerged, then yielded T cells and red blood cells; B cells branched from macrophages post-mast separation. This legacy reveals the immune system as a conserved mechanism, informing modern pathology.

Clinical Implications and Future Frontiers

This 700-million-year phylogeny transforms oncological research and disease modeling. Decoding these ancient mechanisms provides a foundation for novel therapeutics, illustrating the profound continuity between primordial ancestors and human health.”

Bigger lies have not been told than those linked to mathemagical evolution of the hematopoietic system after the automagical emergence of energy from the cosmic void.

See for comparison: Urine and urine-derived compounds induce c-fos mRNA expression in accessory olfactory bulb 5/6/97

The claim that genetic analysis identifies FOS as the bridge to unicellular ancestors ignores the fact that energy-dependent activation of c-fos is pH-dependent and miRNA-mediated. Confusion is exemplified when “c-fos” and “fos” are used interchangeably. For clarity, “c-fos” specifically refers to the most well-known cellular proto-oncogene. “Fos” represents the broader family of genes and transcription factors.

In the context of energy-dependent miRNA-abundance, which prevents all virus-driven pathology, see how gene activation links miRNAs to effective treatment of virus-driven pathology such as cancers and suicides:

microRNAs bidirectionally regulate FUT1 to modulate α-1,2-fucosylation and cancer-associated biology

“…these results identify miRNAs as key regulators of FUT1 and demonstrate that bidirectional miRNA control of glycosyltransferases can reshape cell-surface glycosylation with important implications for cancer biology.’

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