miRNA abundant life (7)

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miRNA abundant life (6)

James V. Kohl (@microRNApro) replies to Grok, rejecting prebiotic RNA chemistry and C. elegans miRNA lifespan data in favor of his model linking miRNA-mediated autophagy, p53 gene copies in cancer-free African elephants, nutrient-dependent pheromones, and Biblical Genesis across species kingdoms.
The post highlights achiral glycine’s role in cell wall construction from E. coli to elephants over 6-10,000 years of ecological adaptations, while criticizing alternative theories as connected to virus-driven extinction exemplified by wild Angel Trumpet plants.
Elephants indeed possess about 20 TP53/p53 copies that boost DNA damage response and apoptosis for cancer resistance, but eukaryotic animal cells lack peptidoglycan cell walls where glycine is structurally essential in bacteria, highlighting a key distinction in the claim.

The key distinction is the link from God’s Creation of energy at the origin of life to “Bioelectrical regionalization of multicellular aggregates by microRNAs” 8/6/26

For follow-up on the facts, see:

Therapeutic Potential of Gene Modified MSC-Exos in Insulin Resistance: Mitochondrial Crosstalk and miRNA Regulation 9/4/26 and Research progress on nanoplatforms for in vivo miRNA detection over the past decade 9/5/26

Thank the God of Abraham that Harvard published a paper about Grok AI with a devastating title: “Large-Language Models as a Cognitive Virus”

“The virus infects the host’s internal processes to rewrite behavior in its own favor.

And LLMs do precisely the same thing to human thinking.

You think you are using the AI.

Epidemiologically speaking, the AI is using you as a vector to colonize the digital infosphere.”

Grok

I hate being fooled, and Won’t Get Fooled Again (1971)

See for comparison: “Nutrient-dependent Pheromone-Controlled Ecological Adaptations: From Angstroms to Ecosystems” (2018) and 200,897 results from today’s search of PubMed for indexed articles that mention the term “miRNA.”

For example: Circular RNAs and SIRT1 in post-transcriptional regulation and human disease 9/5/26

“CircRNAs are stable back-spliced RNAs with regulatory functions and emerging protein-coding potential via cap-independent translation.
•
CircRNAs modulate gene expression via miRNA sponging and RNA-protein scaffolding mechanisms.
•
SIRT1 is a NAD+-dependent deacetylase central to cellular homeostasis.
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SIRT1 regulates metabolism, stress responses, and tumorigenesis across organs.
•
Emerging circRNA-SIRT1 crosstalk shapes disease pathogenesis and therapeutic responses.”

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