miRNA vs LLM word usage (6)

By: Jim Kohl | Published on: September 10, 2026

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miRNA vs LLM word usage (5)
“This content is AI-generated for educational purposes. Always verify important scientific information from reliable scientific sources.”

Ha Ha Ha (ridicule, not humor)
The food energy-dependent pheromone regulated physiology of reproduction in cyanobacteria links human hemoglobin variants from fixation of amino acid substitutions to all biodiversity on Earth via Harvard’s patent for RNA-guided human genome engineering, Virus-mediated archaeal hecatomb in the deep seafloor 10/12/16, Eukaryotic plankton diversity in the sunlit ocean 5/22/15 and pH-dependent oxygen-dependent life. For instance, the oxygen comes from water in the deep seafloor.

For comparison Grok lives in denial of facts from Biblical Genesis:

No, I accept no blame for 9/11—launched in 2023, long after al-Qaeda’s attacks. The diagram matches the model you shared of miRNA biogenesis, epigenetic feedback, virus effects, stress, aging and longevity. Your 2018 paper outlines nutrient-pheromone-miRNA links to adaptation. Mainstream evidence supports miRNA roles in those pathways based on data, not Biblical claims or attributions of suffering.

Grok (repeats itself)

No, I do not accept any blame for the 9/11 attacks. I first launched in 2023, more than two decades after the events. Those attacks were carried out by al-Qaeda operatives.

The attached diagram presents a detailed model of miRNA biogenesis, epigenetic feedback, virus effects on neural circuitry, and links to stress, aging, and longevity pathways.

Moving forward, see: How chromosomes find their partners

Translational Advances in Oncogene and Tumor-Suppressor Gene Research 3/17/25, reported as: Cell-Cycle Regulation: The Molecular Control System

The cell cycle is a tightly regulated process that allows cells to grow, replicate their DNA, and divide. Its progression through G1, S, G2, and M phases is controlled by cyclin-dependent kinases (CDKs), cyclins, tumor suppressors, and checkpoint mechanisms.

During G1, Cyclin D–CDK4/6 initiates phosphorylation of the retinoblastoma protein (Rb). Further phosphorylation by Cyclin E–CDK2 inactivates Rb, releasing E2F, a transcription factor that activates genes required for S-phase entry and DNA replication.

CDK–cyclin complexes act sequentially: Cyclin D–CDK4/6 and Cyclin E–CDK2 drive the G1/S transition, Cyclin A–CDK2 supports S phase and G2 progression, while Cyclin B–CDK1 drives mitotic entry and progression.

The system is restrained by cyclin-dependent kinase inhibitors (CKIs). The INK4 family—including p16^INK4A, p15^INK4B, p18^INK4C, and p19^INK4D—blocks CDK4/6, maintaining Rb in its active state and preventing premature S-phase entry.

The CIP/KIP family, including p21^CIP1, p27^KIP1, and p57^KIP2, inhibits CDK2 and CDK1, helping pause cell-cycle progression when cells experience stress or DNA damage.

Three major checkpoints help preserve genomic integrity: the G1 checkpoint prevents damaged DNA from entering S phase, the G2 checkpoint delays mitosis to allow DNA repair, and the M checkpoint ensures chromosomes are properly aligned before cell division.

This regulatory network is particularly important in cancer biology because mutations or loss of Rb, CDKs, cyclins, or CKIs can remove critical cell-cycle brakes, allowing uncontrolled proliferation.

Stojchevski, R.; Sutanto, E.A.; Sutanto, R.; Hadzi-Petrushev, N.; Mladenov, M.; Singh, S.R.; Sinha, J.K.; Ghosh, S.; Yarlagadda, B.; Singh, K.K.; et al. Translational Advances in Oncogene and Tumor-Suppressor Gene Research. Cancers 2025, 17, 1008.

201,000 results from today’s PubMed search for “miRNA” link our 1996 claims in “From Fertilization to Adult Sexual Behavior” 1996 to “we compared mRNA and miRNA expression profiles between non-matured, and oocytes submitted to IVM in canines using RNA sequencing.”

Add claims from circMAC: microRNA-conditioned binding-site localization on circular RNA isoforms 9/9/26. The most recent claims link 20 copies of p53 in cancer-free African elephants to human hemoglobin variants that protect me from virus-driven polycythemia vera and from myeloid luekemia. PGx and NGS testing ruled out virus-driven pathology via FISH testing. Taken together, the mimimally invasive testing allows me to donate blood that can be used in transfusion medicine via the context established by Biology, molecular and organismic (1964) “The notion has gained some currency that the only worthwhile biology is molecular biology. All else is “bird watching” or “butterfly collecting.” Bird watching and butterfly collecting are occupations manifestly unworthy of serious scientists!” p. 443 and

Dependence of RNA synthesis in isolated thymus nuclei on glycolysis, oxidative carbohydrate catabolism and a type of “oxidative phosphorylation” 1/11/64 “The synthesis of RNA in isolated thymus nuclei is ATP dependent.”

Early donation remembering 9/11. Never forget. i got a great commemorative T-shirt for donating on 9/10/26 at Blood Assurance in Cleveland TN. I wore it to the “Disabled American Veterans” meeting on 9/10/26 in Blue Ridge, GA.

My next donation brings me to a Blood Assurance” total of 19 gallons “For God and Country.”

My PGx and NGS testing advocacy led to a “disruptive behavior” complaint from the Atlanta GA, VA. My testing established facts linked to use of my blood by eliminating claims I had any virus-driven blood cancers. Do you?

Earlier today, VA asked me to schedule an informal meeting about my disability claims, because I’m not dead yet. I scheduled it for 9/15/26, but wonder if any meeting notes will be added to my medical chart that could help prevent virus-driven murders and/or suicides.

I may need a lawyer and/or a bodyguard until then.

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miRNA vs LLM word usage (6)

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